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In nerves uncovered to a hundred Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose additional vitality substrate is required when the tissue vitality demand will increase. Such information are important as they present that glycogen has an vital position below physiological conditions. The fact that glycogen supported CAP conduction within the absence of exogenously utilized glucose in all probability has no worth in ascribing a job to glycogen, since aglycaemia is an artificial experimental procedure, and hypoglycemia is an iatrogenic consequence of insulin therapy that didn't exist prior to 1922 (Frier et al., 2014), thus can't be glycogen’s foremost role. However supplying lactate to axons when tissue power demand increases implies a novel function for mind glycogen and suggests it acts as an vitality buffer quite than an vitality reserve, whose function is not to single-handedly support conduction, however to act in concert with ambient levels of normoglycemic glucose.

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